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Cell Mol Biol 1998, 19:700-708. LeVine AM, Bruno MD, Huelsman KM, Ross GF, Whitsett JA, Korfhagen TR: Surfactant protein A-deficient mice are susceptible to group B streptococcal infection. J Immunol 1997, 158:4336-4340. Li G, Siddiqui J, Hendry M, Akiyama J, Edmondson J, Brown C, et al.: Surfactant protein-A--deficient mice display an exaggerated early inflammatory response to a beta-resistant st
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Ctant protein Adeficient mice. Pneumon 2009, 22:143-155. 35. Hyka N, Dayer JM, Modoux C, Kohno T, Edwards CK III, Roux-Lombard P, et al.: Apolipoprotein A-I inhibits the production of interleukin-1beta and tumor necrosis factor-alpha by blocking contact-mediated activation of monocytes by T lymphocytes. Blood 2001, 97:2381-2389. 36. Groves E, Dart AE, Covarelli V, Caron E: Molecular mechanisms of
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CYP complement and no detectable CYP1-like geneBLAST Searches using CYP1 family protein sequences from various vertebrates and the invertebrates did not result in CYP- like sequences in O. dioica geneome. We then searched the genome for all CYP sequences and unambiguously identified 23 unique genes (Additional file 1, Figure S1, Additional file 2). Thus, Oikopleura seems to have the smallest CYP g
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Eliminate antibodies that did not recognize the antigenic peptide, (3) antibody binding is blocked by incubation with an excess of the antigenic peptide (preadsorption control), (4) Western blots indicate that the primary bands of labeled protein have molecular weights within the range of those previously observed for glycosylated, dimerized or chaperone protein linked angiotensin receptors [68, 1
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CYP complement and no detectable CYP1-like geneBLAST Searches using CYP1 family protein sequences from various vertebrates and the invertebrates did not result in CYP- like sequences in O. dioica geneome. We then searched the genome for all CYP sequences and unambiguously identified 23 unique genes (Additional file 1, Figure S1, Additional file 2). Thus, Oikopleura seems to have the smallest CYP g
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Tta calculations and symmetric docking calculations starting from the CS-Rosetta monomers do not show convergence, indicating an interleaved dimer interface. The fold-and-dock protocol, supplemented by 45 RDCs, converges to a 2.5 ?structure, which shows the correct interleaved backbone topology (Supporting Information Figure 1). Very similar results were obtained for the structural genomics target
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Lear receptor genes. SXR and CAR serve as xenobiotic sensors that activate the transcription of some CYP genes in vertebrates [11,12]. In vertebrates, the NR1H subfamily of nuclear receptors liver-X-receptor (LXR) and farnesoid-X-receptor (FXR) are activated by sterols and bile acids, respectively [29]. LXR and FXR, also found in invertebrates [2], are potential targets of xenobiotic compounds. Ge
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Ve response without requiring multiple booster shoots, thus significantly reducing theVe response without requiring multiple booster shoots, thus significantly reducing the vaccine costs. VLPs do not need attenuation or inactivation - as the live attenuated and killed/ inactivated vaccines - avoiding all the possible side effects of inactivation treatments on the epitope modifications. Moreo

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